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Topics:
Thoracic Malignancies
•
Medical Oncology
Do you take into account ALK fusion variants in your practice for deciding treatment for ALK-NSCLC?
Are there atypical ALK fusion partners that indicate sensitivity to ALK inhibitors?
Related Questions
How are you approaching patients with early-stage NSCLC who progress on neoadjuvant chemo-immunotherapy and are no longer surgical candidates?
What second line therapy do you use for metastatic thymoma that recurs following CAP?
Would you ever consider repeating chemoradiation for patients with locally recurrent small cell lung cancer after prior chemoradiation for LS-SCLC?
How would you approach first-line treatment in metastatic NSCLC for a patient with ALK-EML4 V3a/b variant and MSI-high status?
In cases where EGFR NSCLC has transformed into small-cell lung cancer after treatment with Osimertinib, and with no CNS involvement, is it advisable to continue osimertinib alongside chemotherapy?
What is your approach to obtain tissue diagnosis for a patient with a lung primary and spine metastasis?
In light of recent retrospective data from France on EGFR TKIs use with PPIs showing negative outcomes in NSCLC patients would you consider discontinuing PPIs or changing PPIs to H2 inhibitors?
What is your preferred approach for managing oligoprogressive NSCLC during second-line or later systemic therapy if patient is otherwise responding well at other sites of disease?
Would you add ALK-targeted therapy for patients with EGFR L858R-mutant lung adenocarcinoma who progress on targeted therapy and develop a concomitant STRN-ALK fusion?
How often, if ever, do patients with initially negative targetable mutation workup become positive later in recurrent lung cancer?