Are there instances where TAVR should be considered for patients with moderate AS and HFrEF?

Perhaps this is better asked provocatively; did AS (with less than severely elevated gradients) result in a decline in LVEF, or did moderate AS cause HFrEF? In the absence of an alternate cause of LV dysfunction, AS may be the only causative elephant in the room. Other subtle markers of AS severity, like AV Ca score (> 2000 men / > 1200 women), hemodynamic response to low-level exercise (blunted or hypotensive SBP response) or dobutamine SE increase in transvalvular gradients, may point to the AS severity being worse than moderate.