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Please select the option that best describes you:
Topics:
Rheumatology
•
Vasculitis
How do you approach worsening memory loss in patients with GCA?
Can it be directly related to disease activity or just a consequence of chronic inflammation?
Related Questions
How would you approach the workup of a female patient who has recurrent sinusitis with polyps (biopsy showed active and chronic inflammation) and myocarditis, but negative ANCA and normal eosinophil counts?
How would you approach the evaluation and management of isolated vasculitis with aneurysms involving the segmental hepatic arteries causing hepatic hemorrhage in an otherwise healthy patient in his 80s?
Do you always pursue biopsy confirmation before diagnosing IgA vasculitis?
How would you approach a male in his 60s with bilateral optic perineuritis/neuritis on MRI, steroid-responsive bilateral jaw pain, normal ESR/CRP, negative temporal artery biopsy, and elevated IgG4?
Would you consider anti-IL-5 therapy (mepolizumab or benralizumab) to either prevent or treat the more severe manifestations of eosinophilic granulomatosis with polyangiitis, such as "infiltrative" (eg cardiomyopathy, pulmonary infiltrates, or gastroenteritis) or "vasculitic" (eg neuropathy, palpable purpura, or glomerulonephritis)?
What is your approach to treating IgA nephropathy in patients who also have IgA vasculitis?
What treatment regimen would you recommend for a patient with biopsy-proven giant cell arteritis and diffuse cutaneous systemic sclerosis?
What options are available for patients who require high dose steroids while taking strong CYP4A4 inducers?
How do you differentiate fibromuscular dysplasia from PAN / abdominal vasculitis?
How would you approach management of a patient with mixed cryoglobulinemic vasculitis with predominant skin involvement and no major organ involvement?