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Topics:
Cardiology
•
Cardiac Electrophysiology
How does outflow tract VT differ in management between structurally normal and structurally abnormal hearts ?
Related Questions
Would you consider an ICD for secondary prevention in an otherwise previously healthy adult found to have severe LV systolic dysfunction admitted s/p VF/VT arrest due to profound hypokalemia and hypomagnesemia, or defer implantation given resolution of arrhythmias after correcting electrolyte abnormalities?
What is the most updated consensus regarding the use of pill in the pocket oral anticoagulation in paroxysmal atrial fibrillation, and populations of patients who are most likely to be considered for enrollment in clinical trials?
Is it possible to have first and second degree AV block, either type 1 or 2, on the same EKG strip?
How do you manage asymptomatic non-sustained atrial arrhythmia in patients with single ventricle and Fontan physiology?
Does the presence of diastolic dysfunction guide subsequent pharmacological, pacing and ablative therapies for atrial fibrillation?
How do you determine the appropriate timing and criteria for safely reintroducing AV-nodal blocking agents after initial stabilization and improvement of renal function and hyperkalemia in patients with BRASH syndrome?
What are your typical recommendations for when a patient can return to work following a cardiac arrest, considering the variation in neurological recovery and the potential ramifications based on the type of job?
Based on most current research regarding the more widespread use of class IC antiarrhythmic drugs, what are your prescribing practices in patients with coronary artery disease?
How do you approach agitation management, especially the use of neuroleptics in hospitalized patients with a prolonged QTc?
Would you consider overdrive pacing for recurrent torsades des pointes in a patient without an obvious drug causing QTc prolongation or significant electrolyte abnormality?
