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Topics:
Hematologic Malignancies
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Pediatric Oncology
•
Leukemia
•
ALL
•
Hematology
How would you manage an early isolated CNS relapse in a pediatric patient with Ph positive ALL?
Related Questions
How do you address logistic barriers related to blinatumomab when treating relapsed B-ALL?
Do you check asparaginase levels for all patients receiving receiving E. coli-derived products, or only in certain clinical situations?
How do you incorporate blinatumomab into therapy for a pediatric or AYA patient with isolated CNS relapse of B-ALL, if at all?
Would you offer intensive CNS prophylaxis to Ph negative B-ALL patients who have possible mandibular nerve involvement on MRI face?
What is the preferred approach for an AYA patient with VHR B-ALL with iAMP21 mutation with an isolated early CNS relapse?
Does the presence of asparaginase antibodies on Granger Genetics testing indicate need to switch asparaginase formulations?
In what situations do you use G-CSF for patients undergoing allogeneic HSCT to facilitate engraftment?
How do you manage severe hypertriglyceridemia in the adolescent & young adult population receiving chemotherapy for ALL, in the absence of complications related to hypertriglyceridemia?
What is the role of post-transplant FLT3 inhibition given the results of BMT-CTN 1506 (MORPHO)?
Would you consider post-BMT maintenance therapy for patients with Ph-like ALL with a JAK2 mutation?
