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Topics:
Cardiology
•
Preventive Cardiology
Is there a role for pharmacologic or exercise stress testing for patients with recurrent syncope of unclear etiology?
Related Questions
Would you consider PCSK9 inhibitors for patients with elevated coronary calcium score or coronary calcification for primary prevention in lieu of statins/ezetimibe and/or bempedoic acid?
Do you recommend against use of enteric coated aspirin as opposed to plain aspirin for secondary prevention of stroke or MI?
What are your thoughts on the results from the AQUATIC trial which showed that the addition of aspirin daily + oral anticoagulation in patients > 6 months from PCI and with high atherothrombotic risk was associated with a higher risk of death, MI, stroke, coronary revascularization and acute limb ischemia, compared to oral anticoagulation alone?
How do you counsel patients with metabolic syndrome who decline statin therapy and have low coronary calcium scores regarding their long-term CVD risk?
Should CCTA be considered the diagnostic test of choice in the outpatient evaluation of chest pain?
What should the LDL target be in patients with prediabetes and high lipoprotein (a) with family history of coronary artery disease?
In an asymptomatic patient who has had a routine TTE for non-cardiac reasons, would you order further work-up if there are any WMA or mild LVEF reduction?
What are your preferred lipid-lowering agents and target LDL reduction goal following initiation of therapy for patients with familial hyperlipidemia without underlying CAD?
When would you consider switching to or adding on a PCSK9 inhibitor to lipid-lowering therapy following hospital discharge for acute coronary syndrome, in light of the results of the VICTORION-INCEPTION trial, provided LDL is still not at goal?
Would an exercise treadmill stress test in a patient with known CAD who did not reach 85% of MPHR due to fatigue be considered low-risk based on a DTS of >5, or non-diagnostic?
