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Please select the option that best describes you:
Topics:
Hepatology
•
Genetic Liver Disease
What is your approach to the management of extra-hepatic manifestations/complications of PBC?
For instance, for pruritus, sicca symptoms, metabolic bone disease, dyslipidemia?
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What is your preferred approach to a patient with incidentally found low ceruloplasmin?
What would you your approach to evaluation and monitoring of a patient with elevated AMA and increased immunoglobulins with a low alkaline phosphatase?
How do you differentiate primary from secondary iron overload?
If a patient has a low ceruloplasmin with normal 24 hours urine copper excretion, how would you go about an approach to evaluation of other disorders of copper metabolism as a cause of liver disease?