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Topics:
Cardiology
•
Interventional Cardiology
Would acute-onset thrombocytopenia and concern for active bleeding with platelet count below 50,000 prompt you to hold plavix and/or aspirin following PCI that was done 1-2 weeks ago?
Related Questions
Should CYP2C19 genotype testing be performed in patients presenting with ISR on Plavix?
What is your preferred intervention for diffuse severe ISR involving two layers of stent?
For patients presenting with suspected type 1 NSTEMI and atrial fibrillation on anticoagulation, do you favor triple therapy or SAPT with systemic anticoagulation instead while awaiting LHC?
Do you always give 325mg aspirin if not already loaded with antiplatelets prior to the start of every LHC, even just for diagnostics in the absence of ACS?
Do you recommend SGLT2 inhibitors be held for 3-4 days prior to an elective diagnostic cardiac catheterization or coronary intervention if the patient is made NPO the day of the procedure?
Do you avoid high-potency P2Y12 inhibitors in favor of clopidogrel in patients with atrial fibrillation on a DOAC who undergo PCI?
What is a reasonable protocol for how long to hold warfarin and/or DOACs before cardiac catheterization?
Do you recommend stepwise de-escalation of dual antiplatelet therapy for patients at high risk of bleeding who have undergone drug-coated balloon angioplasty?
Would you favor culprit-only PCI, complete revascularization via percutaneous approach, or urgent CABG evaluation for a young diabetic patient with newly reduced LVEF < 35% presenting with an anterior STEMI and multivessel disease?
What clinical features in suspected ANOCA push you toward proceeding directly to invasive coronary function testing rather than empirically escalating antianginal therapy first?
